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Impaired apolipoprotein synthesis is the most important pathogenetic factor in several types of toxic fatty liver and in the fatty liver produced by protein-calorie malnutrition. Toxic inhibition of protein synthesis can lead to a fatty liver through inhibition of mRNA synthesis or translation. In microvesicular fatty asthma liver, small droplets of triglyceride plus FFA, cholesterol, and phospholipid collect in subcellular organelles. The basic defect is unknown, asthma even though pathologic and clinical features from diverse causes are somewhat similar. asthma The biochemical basis may be a disturbance in the mitochondrial-oxidative pathway, depressing FFA oxidation and impairing apolipoprotein synthesis for VLDL assembly. Fatty liver may result from the accumulation of other neutral lipids. Fat and cholesterol (seen as rhomboid birefringent crystals under polarizing microscopy) are present in Wolman's disease and cholesterol ester storage disease.
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